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Across several preclinical models, Epitalon demonstrated a reduction in chromosomal aberrations, i.e., those misalignments, breaks, and translocations that set the stage for malignant transformation

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ABDH8 was recently discovered to function as a scaffold to attract ZDHHC12 to NLRP3, promoting NLRP3 palmitoylation and the subsequent CMA-mediated degradation of NLRP3, although it lacks an acyltransferase active motif (124)

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